由於取消咗SBA
Paper 1B 嘅重點係實驗題
背書題會少咗好多
實驗題問法有：
（1 ）回應data題
例如：
Describe the trend of ….
Compare curve A to B
呢d題目多數 2-3分 而背書1分都冇
佢想你用文字整合數據
例如俾條curve 你 就想你講幾時rate最高幾時最低 用背書解釋係0分
Compare curves會高分d
頭2分：喺幾分幾秒 A大於B/ B大於A/ A等於B
最後1分：整體趨勢 例如 At all time, A多於B 呢1分成日唔記得寫
（2） 回應data題+ 解釋
例如
Describe and explain…
同（1）最大分別係要講形成呢個結果背後嘅原因係乜嘢 原因又分兩種：
(a) 題目嘅independent variable
(b) 純粹背書吹水
幾時用 a 幾時用b 幾時用a+b 題目會講 例如話 based on the result and your knowledge 就一定要a+b
（3）步驟題
例如
What is the purpose of step 1
If step 1 is skipped…
What is the purpose of adding…
呢題通常2分
第1分： 呢個步驟背後嘅目的
第2分：呢個步驟背後嘅原理
第1分答法 To ensure the DV is due to IV only (萬能key)
第2分答法 睇吓有咩physical / chemical change
例如題目話 study the effect of temperature (IV) on transpiration rate (DV) of plant A
問what is the purpose of adding oil (2分）
第1分 To ensure the change in transpiration rate (DV) is due to temperature (IV) only
第2分 To prevent water evaporation (physical change)
如果第2題問 why use boiled enzyme / why heating
第1分 你懂的
第2分 To denature the enzyme (chemical change)
（4）how 題
例如
How oxygen enters bloodstream
Describe the process of seed germination
背書+推論
由開始到結果 每1步都要講 缺一不可
而邊1步有分 marking scheme 話事
（5）鳩估題
1分就skip咗佢 唔好嘥時間
想我繼續講或者有野問就回覆
冇人回我就收皮
bio dse 2022
Lubishian
360 回覆
23 Like
10 Dislike
more
Lm
Lm
More pls
唔識（1）（2）點做可以做Bio A-Level paper 1b 全部都係問數據描述 但有少少out syll
more pls
2022 bio dser在此
2022 bio dser在此題目分析（1）自創how題
Q: Due to ageing, some people do not produce enough neurotransmitters inside vesicles in the pre-synaptic neuron.
A1: Using your knowledge on neuromuscular junctions and muscle contraction,
explain how people having not enough neurotransmitters may feel that they are lacking muscle strength? (4 marks)
識答可以一齊答
Q: Due to ageing, some people do not produce enough neurotransmitters inside vesicles in the pre-synaptic neuron.
A1: Using your knowledge on neuromuscular junctions and muscle contraction,
explain how people having not enough neurotransmitters may feel that they are lacking muscle strength? (4 marks)
識答可以一齊答
已經寫咗答案 聽日考完中文開波
利申本人re-JUPAS
俾返幅圖先
Q2
If gaseous exchange in the Lung is stopped, how could this failure affect muscular activities? (5 marks)
If gaseous exchange in the Lung is stopped, how could this failure affect muscular activities? (5 marks)
Q3
A gene encoding the muscle receptors has be mutated. The mutated receptors could not generate electrical signals to initiate muscle contraction. Could you explain how the gene mutation results in a malfunction receptor? (4 marks)
A gene encoding the muscle receptors has be mutated. The mutated receptors could not generate electrical signals to initiate muscle contraction. Could you explain how the gene mutation results in a malfunction receptor? (4 marks)
When the motor centre of the cerebral cortex initiate nerve impulses to the motor neurone for muscle contraction, neurotransmitters are released from the axon terminal of the motor neurone into the synapse.
As there are not enough neurotransmitter molecules, even the nerve impulse arrive the presynaptic axon terminal, there will be insufficient neurotransmitter being released from synaptic vesicles into the synaptic cleft by exocytosis, resulting in a lower concentration of neurotransmitter diffusing across the synaptic cleft of the neuromuscular junction.
This lead to less neurotransmitter receptors being stimulated on the postsynaptic muscle fibres. Therefore, less electrical impulse has been generated by the muscle fibre which lead to a weaker contraction force produced as expected and causing the sense of lacking muscle strength.
As there are not enough neurotransmitter molecules, even the nerve impulse arrive the presynaptic axon terminal, there will be insufficient neurotransmitter being released from synaptic vesicles into the synaptic cleft by exocytosis, resulting in a lower concentration of neurotransmitter diffusing across the synaptic cleft of the neuromuscular junction.
This lead to less neurotransmitter receptors being stimulated on the postsynaptic muscle fibres. Therefore, less electrical impulse has been generated by the muscle fibre which lead to a weaker contraction force produced as expected and causing the sense of lacking muscle strength.
Muscular activities require oxygen. The respiration of neurones and muscle cells requires oxygen for ATP generation and the exocytosis of neurotransmitter molecules from the axon terminal to the synapse is an active process which require ATP and oxygen as well.
If gas exchange in lungs is stopped, there will be a lower level of oxygen in blood, there might be damage to the brain cerebral cortex which lead to less motor impulse generation. Also, the low blood oxygen level would lead to lower level of neurones, muscle cells activity and a lower level of neurotransmitter molecules release from synaptic vesicles in the axon terminal into the synaptic cleft.
These leads to a lower level of neurotransmitter receptor stimulation and lower the muscle electrical impulse generation. Thus causing lower muscle activity.
If gas exchange in lungs is stopped, there will be a lower level of oxygen in blood, there might be damage to the brain cerebral cortex which lead to less motor impulse generation. Also, the low blood oxygen level would lead to lower level of neurones, muscle cells activity and a lower level of neurotransmitter molecules release from synaptic vesicles in the axon terminal into the synaptic cleft.
These leads to a lower level of neurotransmitter receptor stimulation and lower the muscle electrical impulse generation. Thus causing lower muscle activity.
Genes are transcribed from DNA to mRNA and translated by tRNA in ribosomes to form proteins in our body.
If the gene code for muscle receptor has been mutated, the base sequence of the particular DNA strand would be altered and different from normal, therefore an abnormal mRNA with defective codons would be transcribed. The defective mRNA will then travel from the cell nucleus to the ribosomes in the cytoplasm or rough endoplasmic reticulum.
As the mRNA has different codons with the normal one, different tRNA with different anticodons might be attracted and bind to the defective mRNA by complementary base pairing, leading to a different amino acid from the normal one being brought to the site. Therefore, the amino acid chain sequence might be altered, lengthened or shortened. And when the amino acid chain is folded and coiled to form a 3D configuration, the polypeptide chain might have be distorted to a abnormal shape and forming a defective receptor, which is protein in nature, with a defective active site that cannot bind by neurotransmitter molecules.
If the gene code for muscle receptor has been mutated, the base sequence of the particular DNA strand would be altered and different from normal, therefore an abnormal mRNA with defective codons would be transcribed. The defective mRNA will then travel from the cell nucleus to the ribosomes in the cytoplasm or rough endoplasmic reticulum.
As the mRNA has different codons with the normal one, different tRNA with different anticodons might be attracted and bind to the defective mRNA by complementary base pairing, leading to a different amino acid from the normal one being brought to the site. Therefore, the amino acid chain sequence might be altered, lengthened or shortened. And when the amino acid chain is folded and coiled to form a 3D configuration, the polypeptide chain might have be distorted to a abnormal shape and forming a defective receptor, which is protein in nature, with a defective active site that cannot bind by neurotransmitter molecules.
唔好自己出題目喇
題目出得唔好，答案都會奇奇怪怪
只會做壞手勢
出Pastpaper已經好夠
題目出得唔好，答案都會奇奇怪怪
只會做壞手勢
出Pastpaper已經好夠
做pastpaper*
肥巴大佬
想講下入考場前建議做咩
1 屙屎
2 同自己講 自己一定得 神就來了
3 心入面講 在座各位都係垃圾
唔建議考場跑圈 傻人發現案
期待相認time 記住官方相認手勢
1 屙屎
2 同自己講 自己一定得 神就來了
3 心入面講 在座各位都係垃圾
唔建議考場跑圈 傻人發現案
期待相認time 記住官方相認手勢
可以參考前幾年嘅post
你個feel有少少痴線…
咁樣發展落去你好容易炒 我肯定
咁樣發展落去你好容易炒 我肯定